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Researcher Database
Name: Hiroshi Kogo
E-Mail address: hkogo@@fujita-hu.ac.jp

Organization / Department (#1)
Organization-1: Fujita Health University
Department(1) 1: Institute for Comprehensive Medical Science
Department(2) 1: Division of Molecular Genetics
Position-1: Assistant Professor
Address-1: 1-98,Toyoake aichi JAPAN

Academic Meeting / Research Meeting (#1)
Academic Meeting-1: The Molecular Biology Society of Japan
Academic Meeting / Research Meeting (#2)
Academic Meeting-2: The Japanese Association of Anatomists
Academic Meeting / Research Meeting (#3)
Academic Meeting-3: The Zoological Society of Japan

Research History [1995] (#2)
Papers-2: 1. Kogo, H., Kudo, A., Park, M.K., Mori, T., Kawashima, S. (1995) In situ detection of gonadotropin-releasing hormone (GnRH) receptor mRNA expression in the rat ovarian follicles. J. Exp. Zool. 272, 62-68.
2. Park, M.K., Kogo H., Kawashima, S., Wakabayashi, K. (1995) Characterization of gonadotropin-releasing hormone (GnRH)-immunoreactive protein in the rat pineal gland. J. Neurosci. Res. 41, 386-393.
Research History [1997] (#4)
Papers-4: 1. Kogo, H., Shioya, M., Takahashi, Y., Fujimoto, T. (1997) Caveolae and endoplasmic reticulum: Immunofluorescence microscopy and time lapse analysis. Acta Histochem. Cytochem. 30, 593-599.
Research History [1998] (#5)
Papers-5: 1. Isshiki, M., Ando, J., Korenaga, R., Kogo, H., Fujimoto, T., Fujita, T., Kamiya, A. (1998) Endothelial Ca2+ waves preferentially originate at specific loci in caveolin-rich cell edges. Proc. Natl. Acad. Sci. USA 95, 5009-5014.
2. Fujimoto, T., Hagiwara, H., Aoki, T., Kogo, H., Nomura, R. (1998) Caveolae: from a morphological point of view. J. Electron Microsc. 5, 451-460.
Research History [1999] (#6)
Papers-6: 1. Kogo, H., Fujimoto, T., Mori, T. (1999) Evidence for gonadotropin-releasing hormone receptor mRNA expression by estrogen in rat granulosa cells. Cell Tissue Res. 297, 459-465.
2. Kogo, H., Fujimoto, T., Park, M.K., Mori, T. (1999) Gonadotropin-releasing hormone (GnRH) receptor mRNA expression in the ovaries of neonatal and adult rats. Cells Tissues Organs. 164, 14-22.
Research History [2001] (#8)
Papers-8: Fujimoto, T., H. Kogo, K. Ishiguro, K. Tauchi and R. Nomura: Caveolin-2 is targeted to lipid droplets, a new ‘membrane domain’ in the cell. J. Cell Biol. 152: 1079-1085, 2001.
Research History [2002] (#9)
Papers-9: Kogo, H., K. Ishiguro S. Kuwaki and T. Fujimoto: Identification of a splice variant of mouse caveolin-2 mRNA encoding an isoform lacking the C-terminal cytoplasmic domain. Arch. Biochem. Biophys. 401: 108-114, 2002.
Research History [2003] (#10)
Papers-10: Aoki, T., S. Kogure, H. Kogo, M. Hayashi, Y. Ohno-Iwashita and T. Fujimoto: Sequestration of cross-linked membrane molecules to caveolae in two different pathways. Acta Histochem. Cytochem. 36: 165-171, 2003.
Research History [2004] (#11)
Papers-11: 1. Kogo, H., T. Aiba and T. Fujimoto: Cell type-specific occurrence of caveolin-1α and -1β in the lung caused by expression of distinct mRNAs. J. Biol. Chem. 279: 25574-25581, 2004.
2. Fujita, Y., S. Maruyama, H. Kogo, S. Matsuo and T. Fujimoto: Caveolin-1 in mesangial cells suppresses MAP kinase activation and cell proliferation induced by bFGF and PDGF. Kidney Int. 66: 1794-1804, 2004.
3. Nakamura, N., T. Akashi, T. Taneda, H. Kogo, A. Kikuchi and T. Fujimoto: ADRP is dissociated from lipid droplets by ARF1-dependent mechanism. Biochem. Biophys. Res. Commun. 322: 957-965, 2004.
4. Kuwahara, M., T. Ogaeri, R. Matsuura, H. Kogo, T. Fujimoto and S. Torihashi: In vitro organogenesis of gut-like structures from mouse embryonic stem cells. Neurogastroenterol. Motil. 16 (Suppl 1): 14-18, 2004.
Research History [2005] (#12)
Papers-12: Inagaki, H., Ohye, T., Kogo, H., Yamada, K., Kowa, H., Shaikh, T.H., Emanuel, B.S., Kurahashi, H. (2005). A palindromic AT-rich repeat in the NF1 gene is hypervariable in humans and evolutionarily conserved among primates. Hum. Mutat., 26, 332-342.

Research History [2006] (#13)
Papers-13: 1. Kogo, H., Ito, S., Moritoki, Y., Kurahashi, H., Fujimoto, T. (2006) Differential expression of caveolin-3 in mouse smooth muscle cells in vivo. Cell Tissue Res., 324, 291-300.
2. Matsuura, R., Kogo, H., Ogaeri, T., Miwa, T., Kuwahara, M., Kanai, Y., Nakagawa, T., Kuroiwa, A., Fujimoto, T., Torihashi, S. (2006) Crucial transcription factors in endoderm and embryonic gut development are expressed in gut-like structures from mouse ES cells. Stem Cells, 24, 624-630.
3. Kato, T., Inagaki, H., Yamada, K., Kogo, H., Ohye, T., Kowa, H., Nagaoka, K., Taniguchi, M., Emanuel, B.S., Kurahashi, H. (2006) Genetic variation affects de novo translocation frequency. Science, 311, 971.
4. Kurahashi, H., Inagaki, H., Ohye, T., Kogo, H., Kato, T, Emanuel, B.S. (2006) Palindrome-mediated chromosomal translocations in humans. DNA Repair, 5, 1136-1145.
5. Kurahashi, H., Inagaki, H., Ohye, T., Kogo, H., Kato, T., Emanuel, B.S. (2006) Chromosomal translocations mediated by palindromic DNA. Cell Cycle, 5, 1297-1303.
Speaker-13: 1. Kogo, H., Ogawa, A., Kowa, H., Yamada, K., Taniguchi, M., Ohye, T., Inagaki, H., Toda, T., Kurahashi, H. (2006) Identification and characterization of meiosis-specific, HORMA domain-containing proteins, HORMAD1 and HORMAD2, as candidates involved in meiotic chromosome behavior in mammals. 20th International Congress of Biochemistry and Molecular Biology, Kyoto, Japan, June, 18-23.
2. Kogo, H., Kowa, H., Yamada, K., Taniguchi, M., Ohye, T., Inagaki, H., Toda, T., Kurahashi, H. (2006) A screen for genes involved in meiosis-specific chromosome behavior based on expression profiling. 11th International Congress of Human Genetics, Brisbane, Australia, August, 6-10.
Research History [2007] (#14)
Papers-14: 1. Kogo, H., Inagaki, H., Ohye, T., Kato, T., Emanuel, B.S., Kurahashi, H. (2007) Cruciform extrusion propensity of human translocation-mediating palindromic AT-rich repeats. Nucleic Acids Res., 35, 1198-1208.
2. Kurahashi, H., Inagaki, H., Hosoba, E., Kato, T., Ohye, T., Kogo, H., Emanuel, B.S. (2007) Molecular cloning of a translocation breakpoint hotspot in 22q11. Genome Res., 17, 461-469.
3. Kato, T., Yamada, K., Inagaki, H., Kogo, H., Ohye, T., Emanuel, B.S., Kurahashi, H. (2007) Age has no effect on de novo constitutional t(11;22) translocation frequency in sperm. Fertil. Steril., 88, 1446-1448.
4. Pryor-Koishi, K., Nishizawa, H., Kato, T., Kogo, H., Murakami, T., Tsuchida, K., Kurahashi, H., Udagawa, Y. (2007) Overproduction of the follistatin-related gene protein in the placenta and maternal serum of patients with pre-eclampsia. BJOG, 114, 1128-1137.
Research History [2008] (#15)
Papers-15: 1. Kato, T., Inagaki, H., Kogo, H., Ohye, T., Yamada, K., Emanuel, B.S., Kurahashi, H. (2008) Two different forms of palindrome resolution in the human genome: deletion or translocation. Hum. Mol. Genet., 17, 1184-1191.
2. Nishizawa, H., Pryor-Koishi, K., Suzuki, M., Kato, T., Kogo, H., Sekiya, T., Kurahashi, H., Udagawa, Y. (2008) Increased levels of pregnancy-associated plasma protein-A2 in the serum of pre-eclamptic patients. Mol. Hum. Reprod., 14, 595-602.
Research History [2009] (#16)
Papers-16: 1. Bolor, H., Mori, T., Nishiyama, S., Ito, Y., Hosoba, E., Inagaki, H., Kogo, H., Ohye, T., Tsutsumi, M., Kato, T., Tong, M., Nishizawa, H., Pryor-Koishi, K., Kitaoka, E., Sawada, T., Nishiyama, Y., Udagawa, Y., Kurahashi, H. (2009). Mutations of the SYCP3 gene in women with recurrent pregnancy loss. Am. J. Hum. Genet., 84, 14-20.
2. Inagaki, H., Ohye, T., Kogo, H., Kato, T., Bolor, H., Taniguchi, M., Shaikh, T.H., Emanuel, B.S., Kurahashi, H. (2009). Chromosomal instability mediated by non-B DNA: Cruciform conformation and not DNA sequence is responsible for recurrent translocation in humans. Genome Res., 19, 191-198.
3. Kurahashi, H. Inagaki, H., Kato, T., Hosoba, E., Kogo, H., Ohye, T., Tsutsumi, M., Bolor, H., Tong, M., Emanuel, B.S. (2009). Impaired DNA replication prompts deletions within palindromic sequences, but does not induce translocations in human cells. Hum. Mol. Genet., 18, 3397–3406.
4. Kurahashi, H., Bolor, H., Kato, T., Kogo, H., Tsutsumi, M., Inagaki, H., Ohye, T. (2009). Recent advance in our understanding of the molecular nature of chromosomal abnormalities. J. Hum. Genet., 54, 253–260.
Research History [2010] (#17)
Papers-17: 1. Kogo, H., Kowa-Sugiyama, H., Yamada, K., Bolor, H., Tsutsumi, M., Ohye, T., Inagaki, H., Taniguchi, M., Toda, T., Kurahashi, H. (2010). Screening of genes involved in chromosome segregation during meiosis I: towards the identification of genes responsible for infertility in humans. J. Hum. Genet., 55, 293-299.
2. Ohye, T., Inagaki, H., Kogo, H., Tsutsumi, M., Kato, T., Tong, M., Macville, M.V., Medne, L., Zackai, E.H., Emanuel, B.S., Kurahashi, H. (2010). Paternal origin of the de novo constitutional t(11;22)(q23;q11). Eur. J. Hum. Genet., 18, 783-787.
3. Kurahashi, H., Inagaki, H., Ohye, T., Kogo, H., Tsutsumi, M., Kato, T., Tong, M., Emanuel, B.S. (2010). The constitutional t(11;22): implications for a novel mechanism responsible for gross chromosomal rearrangements. Clin.Genet., 78, 299-309.
4. Tong, M., Kato, T., Yamada, K., Inagaki, H., Kogo, H., Ohye, T., Tsutsumi, M., Wang, J., Emanuel, B.S., Kurahashi, H. (2010). Polymorphisms of the 22q11.2 breakpoint region influence the frequency of de novo constitutional t(11;22)s in sperm. Hum. Mol. Genet., 19, 2630-2637.

Sat Nov 20 07:25:15 JST 2010

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